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By K. Stejnar. University of the Sciences in Philadelphia.

A comparison of four models of total knee replacement prostheses generic imdur 60 mg amex. A longitudinal study of the radiolucent line at the bone–ce- ment interface following total joint replacement procedures purchase 30mg imdur amex. Loosening of the cemented acetabular cup in total hip replacement. Reaction of bone to methacrylate after hip arthroplasty. An experimental study in the rabbit using the bone growth chamber. Monomer leakage from polymerizing acrylic bone cement. An in vitro study on the influence of speed and duration of mixing, cement volume and surface area. Acute local tissue effects of polymerizing acrylic bone cement. An intravital microscopic study in the hamster’s cheek pouch on the chemically induced microvascular changes. A comparative study in the rabbit’s ear on the toxicity of methyl methacrylate monomer of varying composition. Effects of polymerization heat and monomers from acrylic cement on canine bone. Bone marrow pressure chamber: a permanently inserted titanium implant for intramedullary pressure measurements. Removal torque for bone-cement and titanium screws implanted in rabbits. Bone reactions to intramedullary insertion of methylmethacrylate. Rhinelander F W, Nelson C L, Stewart R D, Stewart B S.

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Am J Knee phy in evaluating the patellofemoral joint before and Surg 1997 cheap 60mg imdur amex; 10: 221–227 order imdur 20 mg on line. Fithian and Eiki Nomura Abstract tainty is justified. Perhaps we’ve been missing Acute patellar dislocation is a common injury something. In the past 10 years, patellofemoral instability and anterior knee research has begun to focus on the injuries asso- pain, there was until recently little attention ciated with acute patellar dislocation, and the given to the structures that are injured during specific contributions the injured structures patellar dislocation, and the contributions these make to patellar stability in intact knees. The injured structures make in controlling patellar implication is that injury to specific structures motion in the intact knee. Since the early 1990s, may have important consequences in convert- some investigators have focused on the individ- ing a previously asymptomatic, though perhaps ual components of the knee extensor mecha- abnormal, patellofemoral joint into one that is nism that limit lateral patellar motion. These studies have vivo studies of the surgical pathology36-43 and been intended to improve the precision of sur- magnetic resonance (MR) imaging studies36,41-44 gical treatment for patellar instability, and their have reported the pathoanatomy of the primary results are driving refinements in our surgical dislocation with specific attention to injuries indications as well as technique. The Introduction importance of these lines of research is that they Patellar dislocation can lead to disabling seque- have focused attention on (1) the pathological lae such as pain and recurrent instability, par- anatomy of the initial dislocation event, and (2) ticularly in young athletes. This represents a novel prevention of recurrent patellar instability after approach to the clinical problem of the unstable the initial dislocation. The purpose of this article is to bring appropriate treatment. Widespread reports of the results and implications of this body of mixed results9,13,19-27 or outright failure11,12 research into perspective within the context of of surgical treatment suggest that such uncer- the prevailing literature on patellar dislocation. Warren and Marshall,47 Kaplan,48 Reider,49 and These components are: (1) bony constraint due Terry. The Layer 1 includes the superficial medial retinacu- combination of articular buttress and soft tissue lum (SMR), which courses from the anterome- tension determines the limits of passive patellar dial tibia and extends proximally to blend with displacement. The medial patellotibial liga- the patellofemoral joint between 30 and 100 ment (MPTL) is an obliquely oriented band of degrees of knee flexion, Ahmed29 reported that fibers coursing from the anteromedial tibia and mediolateral patellar translation was controlled blending with the fibers of the retinaculum to by the passive restraint provided by the topo- insert on the medial border of the patella. In particular, patellar medial-lateral patellofemoral ligament (MPFL), along with the translation was controlled by the trochlear superficial medial collateral ligament (MCL), to topography, while retropatellar topography also be part of layer 2.